Protein Wnt-7a, ELISA Kit

Protein Wnt-7a; Wnt7a; Wnt-7a

For Research Use Only. Not for use in diagnostic procedures.

For long term storage, please store the entire kit at -20 degree C.

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Small volumes of Wnt7a elisa kit vial(s) may occasionally become entrapped in the seal of the product vial during shipment and storage. If necessary, briefly centrifuge the vial on a tabletop centrifuge to dislodge any liquid in the container`s cap. Certain products may require to ship with dry ice and additional dry ice fee may apply.

MBS2884712 is a ready-to-use microwell, strip plate ELISA (enzyme-linked immunosorbent assay) Kit for analyzing the presence of the Protein Wnt-7a (Wnt7a) ELISA Kit target analytes in biological samples. The concentration gradients of the kit standards or positive controls render a theoretical kit detection range in biological research samples containing Wnt7a. The ELISA analytical biochemical technique of the MBS2884712 kit is based on Wnt7a antibody-Wnt7a antigen interactions (immunosorbency) and an HRP colorimetric detection system to detect Wnt7a antigen targets in samples. The ELISA Kit is designed to detect native, not recombinant, Wnt7a. Appropriate sample types may include undiluted body fluids and/or tissue homogenates, secretions. Quality control assays assessing reproducibility identified the intra-assay CV (%) and inter-assay CV(%).

38,974 Da

wingless-type MMTV integration site family, member 7A

protein Wnt-7a

Protein Wnt-7a

Wnt-7a  [Similar Products]

WNT7A_MOUSE

WNT7A: Ligand for members of the frizzled family of seven transmembrane receptors. Probable developmental protein. Signaling by Wnt-7a allows sexually dimorphic development of the mullerian ducts. Defects in WNT7A are the cause of limb pelvis hypoplasia aplasia syndrome (LPHAS). A syndrome of severe deficiency of the extremities due to hypo- or aplasia of one or more long bones of one or more limbs. Pelvic manifestations include hip dislocation, hypoplastic iliac bone and aplastic pubic bones. Thoracic deformity, unusual facies and genitourinary anomalies can be present. Defects in WNT7A are a cause of Fuhrmann syndrome (FUHRS); also known as fibular aplasia or hypoplasia femoral bowing and poly- syn- and oligodactyly. Fuhrmann syndrome is a distinct limb-malformation disorder characterized also by various degrees of limb aplasia/hypoplasia and joint dysplasia. Belongs to the Wnt family.

Protein type: Secreted, signal peptide; Secreted

Cellular Component: cell surface; extracellular region; extracellular space; proteinaceous extracellular matrix

Molecular Function: cytokine activity; frizzled binding; protein binding; receptor agonist activity; receptor binding

Biological Process: angiogenesis; asymmetric protein localization; cartilage condensation; cartilage development; cell fate commitment; cell proliferation in forebrain; cell-cell signaling; central nervous system vasculogenesis; cerebellar granule cell differentiation; chondrocyte differentiation; dorsal/ventral pattern formation; embryonic axis specification; embryonic digit morphogenesis; embryonic forelimb morphogenesis; embryonic hindlimb morphogenesis; embryonic limb morphogenesis; establishment of cell polarity; fallopian tube development; forelimb morphogenesis; hindlimb morphogenesis; limb development; multicellular organismal development; negative regulation of apoptosis; negative regulation of neurogenesis; neurotransmitter secretion; organ morphogenesis; palate development; positive regulation of cell proliferation; positive regulation of epithelial cell proliferation involved in wound healing; positive regulation of JNK cascade; positive regulation of synaptogenesis; positive regulation of transcription from RNA polymerase II promoter; positive regulation of transcription, DNA-dependent; regulation of axon diameter; regulation of axonogenesis; regulation of cell proliferation; reproductive structure development; response to estrogen stimulus; satellite cell activation; satellite cell compartment self-renewal involved in skeletal muscle regeneration; signal transduction; skeletal morphogenesis; skin morphogenesis; somatic stem cell division; somatic stem cell maintenance; stem cell development; synapse organization and biogenesis; uterus development; Wnt receptor signaling pathway; Wnt receptor signaling pathway through beta-catenin

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